This product is a box of 10 vials.
GHRP 2 (otherwise called KP 102) is an engineered hexapeptide Development Hormone Discharging Peptide (GHRP), which follows up on the hypothalamus and the pituitary organ to discharge development hormone with a slight stimulator impact on Prolactin, ACTH and Cortisol levels. GHRP 2 is thought to be a genuine hGH secretagogue, implying that it invigorates the body’s own emission of hGH. Human Development hormone has been appeared in studies to advance incline body mass and decrease adiposity (fat). GHRP 2 has exhibited that it is exceptionally compelling at fortifying GH creation in exploration test subjects. It has a short half existence with top focuses happening around 15 minutes and not longer than a hour after organization.
What Is GHRP-2 ?
GHRP-2 (also known as pralmorelin) is a synthetic growth hormone secretagogue known to bind to the ghrelin/growth hormone secretagogue receptor. It was the first of the growth hormone secretagogues to be introduced and is currently marketed as a test peptide for assessing growth hormone deficiency and secondary adrenal failure. GHRP-2 has been investigated in stage II clinical trials for evaluating short stature and is under active research for its effects on appetite, muscle growth, the immune system, and sleep cycles. GHRP-2 is orally and sublingually active, so it does not have to be injected to have its effects.
Sequence: D-Ala-D-2Nal-Ala-Trp-D-Phe-Lys
Molecular Formula: C45H55N9O6
Molecular Weight: 817.9749 g/mol
PubChem CID: 6918245
CAS Number: 158861-67-7
Code Names: KP-102, GPA-748, WAY-GPA-748
Muscle Structure
Research in yaks indicates that GHRP-2 can boost muscle growth in two ways: increased protein deposition and decreased protein degradation. The research showed that GHRP-2 can overcome natural growth limits that occur in yaks due to food shortages, disease, and harsh environmental conditions like cold. The most important finding from this study is that GHRP-2 reduces muscle wasting by inactivating atrogin-1 and MuRF1, proteins that control the muscle breakdown pathway. There is hope that these findings could be used to reduce the muscle loss common in chronic illnesses like autoimmune disease, cancer, and more.
By activating growth hormone and insulin-like growth factor-1, GHRP-2 helps to boost muscle protein deposition. The combined effect of reducing breakdown and enhancing deposition is that GHRP-2 encourages the development of lean body mass even in tough conditions.
Stimulates Appetite
GHRP-2 has been shown to boost food intake. Though this may not seem immediately important, stimulating appetite in cases of chronic disease is a key part of overall health care. The ability to easily and reliably stimulate appetite could help doctors treat patients with long-term illnesses and improve outcomes over time.
May Protect the Heart
Research in fetal heart cell cultures shows that GHRP-2 and its similar versions (GHRP-1 and GHRP-6) can help protect heart cells by reducing apoptosis, or programmed cell death. This is especially important after a heart attack, when heart cells are more likely to die due to reduced blood and nutrient supply. Research using a similar version of GHRP-2 called Hexarelin has suggested there may be a specific receptor for these peptides. Identifying new receptors in any tissue opens paths for new drug development, but also deepens our understanding of human physiology and how to prevent problems in the first place.
Improves the Immune System
GHRP-2 has been shown to stimulate the thymus, an organ responsible for protecting certain cells of the immune system. In particular, the thymus helps T cells to mature. T cells are critical for adaptive immunity and our ability to fight off complex infections. Function of the thymus declines with age, which leads to a number of age-related issues ranging from poor tissue repair to lost immunity and thus an inability to fight off infections, guard against cancer, and maintain normal tissue function. GHRP-2 has been shown to rejuvenate the thymus, boosting the number and variety of T-cells it produces. This leads to improved immunity.
Improves Sleep Quality
GHRP-2 has been shown to increase the duration of stages 3 and 4 of the sleep cycles by about 50% each while increasing REM sleep by approximately 20% and reducing the amount that a person deviates from “normal sleep” by as much as a third. Overall, the improvement in sleep led to better cognitive function, blood pressure, healing, and energy levels. These findings, while useful to all adults, are particularly important in older people, where aging takes a toll on sleep quality. GHRP-2 may be useful in understanding how to fine-tune sleep to improve quality and maybe even help people get all the benefits of a full night’s sleep in less time.
Pain Perception
It was originally thought that observations of decreased pain in animal models of osteoarthritis were the result of GHRP-2 boosting growth hormone levels and speeding up healing in damaged tissues. Keen scientists observed, however, that the pain relief happened well before healing, suggesting that GHRP-2 may have direct effects on pain perception. As it turns out, GHRP-2 has action at the opioid receptors.
There are four known opioid receptors. Most opioid pain medications do not tell them apart. This is problematic because while some receptors handle pain, others affect things like wakefulness and breathing while still others impact addiction. GHRP-2 is a selective opioid receptor activator, binding mainly to the receptors responsible for pain relief, sedation, and addiction. This finding indicates that it may be possible to create selective opioid activators and therefore reduce or prevent entirely unwanted effects like slowed breathing and addiction.
GHRP-2 exhibits minimal to moderate side effects, low oral and excellent subcutaneous bioavailability in mice. Per kg dosage in mice does not scale to humans. GHRP-2 for sale at Peptide Sciences is limited to educational and scientific research only, not for human consumption. Only buy GHRP-2 if you are a licensed researcher.
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